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74 – 76 If the leading PD post-synaptic damage cause is neurotoxins and glutathione is the most powerful protective mechanism, then administrating adequate glutathione precursors at proper levels will positively impact the clinical course
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Regarding hepatic pathological setting, mitochondrial glutathione plays a critical role in the survival of hepatocytes during hypoxia through the regulation of mitochondrial generation of oxidative stress [71]
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